Dealing with Clostridium difficile.

Written by Lee Stevenson, sorry I am not the best editor.

This is not medical advice I have written this for educational purposes.

Clostridium infection is preventable unless you rely heavily on modern medicine because it is one of the number one causes. Everyone has Clostridium defficile in their guts so to call it a pathogen is inaccurate. We need to determine what went wrong to make it become overgrown.

Proton pump inhibitors, antihistamines and antibiotics increases the risk of c diffe. Low stomach acid causes c diffe overgrowth. Low digestive enzyme production and low stomach acid production causes C diff overgrowth. Using trimethylglycine can increase stomach acid. Dimethylglycine should not be used with C diff because it increases dopamine. Protein from meat , sialic acid and glucose feeds C diffe. The para-cresol that Clostridium produces inhibits other microbes from growing so a person should restore the microbes that break down cresol first so that other commensals can reestablish. Clostridium difficile is not very good at competing for nutrients so is kept in check by other gut microbes. Once antibiotics, chemicals in our foods like , or a loss of stomach acid production causes a loss of competing microbes it can get established. This is common with many microbes that are a normal part of the gut flora. Candida can also become infectious when the stomach becomes too alkaline. The inflammation in the gut is from high TH17 cells they should not be reduced , it is a sign the body has detected it and is trying to get rid of it. Focus on restoring gut PH and pancreatic enzymes. A person with C diffe may need to supplement with digestive enzymes to help restore proper gut PH until the commensal microbes are restored. Once the commensals are restored it should not be necessary to inhibit TH17 because the commensal gut microbes will inhibit it. Taking Saccharomyces boulardii has been found to dramatically improve success of getting C diff levels down enough to restore normal microbiome. Many Bacillus strains produce natural antibiotics that inhibit C diffe. This is because C diff is in the soil and Bacillus is found in the soil and the Bacillus prevents competition of the environment with C diff by producing natural antibiotics against C diff. The body produces Pyrrole-2-carboxylate to fight infection in the gut and urinary system. C diff produces a toxin that inhibits DAO enzymes used to produce this metabolite from being produced. When we lose the microbes that produce secondary bile acid taurocholic acid levels increase and this feeds C diff. The also inhibits FXR which farther inhibits bile production which would cause malabsorption and farther C diff growth. Secondary bile prevents the toxic effects of the metabolites that C diff produces. In order to restore the microbiota that maintain secondary bile acids it will be necessary to use things such as digestive enzymes and other things to help increase the PH of the digestive tract so the commensal microbes can get established. Once the secondary bile acid are restored the body will detox the toxic metabolites produced that caused the high dopamine levels and the brain will start to heal. It does take a very long time for the brain to heal so do not expect things to happen quickly. Deoxycholic acid is the most potent metabolite produced by our gut microbes against C diff.
C diff also increases NADPH oxidase activity which increases oxidative stress and can inhibit the genes needed for beta oxidation which can lead to obesity, muscle fatigue, and low energy levels. The oxidative stress is very damaging to the body.

Until the secondary bile acid producer are restored it may be necessary to use FXR agonist because when FXR is inhibited it causes fatty liver disease. This also causes a person to develop diabetes because the develop insulin resistance. Increasing FXR expression has been found to help restore bile homeostasis. The inhibited bile production also causes commensals that usually provide nutrients for the body to become inflammatory. Taurocholic acid levels when high kills some of the secondary bile acid producers so activated charcoal may be needed to help reduce TCA to help the beneficial microbes get established. Bear bile activates FXR it has been shown to neutralize the toxins produces by C diff.

The commensal Clostridium bacteria produce secondary bile acid that prevents C diff overgrowth. Hydrolyzed protein helps to increase the commensal Clostridium. Eubacterium species also produces deoxycholic acid. Some Bacteroides and commensal Escherichia produce secondary bile acid. None digestable fiber increases most Commensal secondary bile acid producers and it also absorbs and removes toxic bile from the body. Polyphenols help to increase the commensals that produce secondary bile acids. Fruit pectin increases the bacteroides that produces secondary bile acids. Besides using hydrolyzed protein fiber also increases commensal Clostridium. So to summarize hydrolyzed protein increases bile and inulin increases the microbes that convert it to secondary bile acid which reduces the bad microbes and increases the good. DCA they type of bile produced by the secondary bile acid producers make reduces inflammation and improves nutrient absorption. C diff is not the only infection that causes inhibited bile production and increases taurocholic acid. So this may help with other types of gut infections.

Until the secondary bile acid producers are restored a person will not be able to absorb nutrients. Things like vitamin B12 may have to be taken in sublinqual form. Also cholesterol and fats are needed to produce bile but with the inhibited digestive enzymes the body cannot properly absorb them so a digestive enzyme with lipase would be needed so the cholesterol and fats needed to produce bile could be absorbed. Sulfation is needed to produce bile so I would also support sulfation. Digestive enzymes with meals will help with the nutrient malabsorption.

Because C diff causes many types of food intolerance they usually recommend avoiding certain foods. I believe this is not necessary if a person takes digestive enzymes when they eat. The only food I would avoid is protein and animal fats because they do feed C diff. It would probably be better to get the fats from things such as olive oil and avocado oil. Foods that are high in luecine and isoleucine should be avoided because those are proteins C diff prefers. So if a person uses hydrolyzed protein they should try to find some they does not contain those proteins. Glucose and fructose are also substrates C diffe can use when protein is not available. Bile can be produced form glycine or taurine. Taurine should be avoid because studies have shown that taurine can increase C difficile. I mentioned earlier sulfation should be supported. I have since learned many pathogens and gut dysbiosis does inhibit sulfation.

Probiotics that have been the most effective at reducing C diffe are Saccharomyces boulardii , Bacillus coagulans, and Lactobacillus rhamnosus GG. Also the microbes that produce valerate are the most effective at knocking C diff down.

Clostridium scindens is the most effective against C diff but creates it’s own problems because it causes testosterone levels to become too high in men and women. So I have been undecided as to whether it would be a good microbe because high testosterone can cause many problems.

Clostridium and other infection cause high acrolein levels. It is also a contaminant that is found in high levels in the environment. Antioxidants especially ferulic acid prevents the toxic effects of acrolein. Acrolein can also formed from damage in the body especially neurological. Some other potent antioxidants found to prevent the toxic effects of acrolein are resveratrol, citrus extract, and curcumin. Ferulic acid is found in pectin, olive oil, spices, walnuts, black beans, crushed flax seed, popcorn, navy beans, rye bread, angelica, cooked sweet corn, cauliflower, tomato, and horse gram are some of the highest sources. It is also produced by our microbiome when we consume liquid chlorophyll. Acrolein causes glycosaminoglycan production to be inhibited. This can cause the blood brain barrier to leak and cause damage to the GAG layers in the bladder and intestine. N-acetylcystein and a glucosamine/chondroitin complex can help prevent that from happening. Acrolein causes excess blood clotting so it may be a good idea to take supplements to address the excess blood clotting.
The research on acrolein is contradicting the body produces acrolein and so do our microbiome and it fights some types of infection. The body also produces it to stimulate healing. So I believe it is involved in protecting and healing the body but causes adverse effects when levels get too high. Restoring normal microbiome restores the ability of the body to detox excess acrolein and it reduces the microbes that produce it.

Almost all short chain fatty acid producers prevent C diff overgrowth but they cannot get established without secondary bile acids so it is very important to get secondary bile acid levels restored. Aribinogalactin increases almost all the microbes that inhibit C diff overgrowth. Pectin also increases many of the microbes that protect for C diff overgrowth. Beans and inulin also increase the microbes that inhibit C diff. Burdock root , pomegranate and grape seed extract also increase the microbes that inhibit C diffe. The other species such as Bifidobacterium and Lactobacillus break down taurocholic acid which make less available for C diff which it uses to enhance it’s growth. Berberine found in bayberry, bearberry and coptis increases the microbes that reduce C diff by activating the FXR. The best digestive enzymes to get would be a complete plant based digestive enzyme that contains lipase because it improves the absorption of fats, cholesterol and fat soluble vitamins needed for the production of bile acids.

Eubacterium upregulates FXR expression also. Low dose milk thistle and low dose chicory root increases FXR expression.

Valerate produced by certain gut microbes inhibits D diff overgrowth. Crushed flax seed and walnuts can help to restore the valerate producing bacteria.

Commensal clostridium prevents PCOS because it is part of our estrobiome. It helps to break down phytoestrogens in our food , converting them to metabolites that help to normalize hormones. If pathogenic Clostridium overgrowth is not addressed it can result in the intestine tissue becoming necrotic requiring it to be cut out and if not addressed in time it can result in death.

If you are deficient in commensal Clostridium you will develop high estrogen levels because it helps reduce estrogen and xenobiotics. You will get constipated because the deficiency in secondary bile acids will cause issues with serotonin. This will cause high 5-HT levels which will inhibit gut motility and make you sensitive to things that increase serotonin. This can cause high anxiety and stress levels.
We need certain microbes in order to be able to convert the phytoestrogens. Barley sprouts help to increase the microbes that convert the phytoestrogens. Eubacterium Limosum, certain Clostridium species are needed to convert phytoestrogens. E. Limosum requires acetate and antibiotics and glyphosate usually kill off the acetate producing microbes from our guts. Studies claim only 30% of the population contain the microbes needed to convert phytoestrogens so the body can use them. There is still benefits from phytoestrogens but they are not as effective if we are lacking the microbes that break them down for our bodies. Over activation of Ahr will cause low estrogen levels so homeostasis is the key.
Commensal Clostridium produces vitamin K and secondary bile acids which activate the FXR receptor. Those with IBD-C are frequently constipated. I noticed a pattern , those with IBD-C are deficient in secondary bile acids and Clostridia commensals which produce secondary bile acids. Fat, carbohydrates and meat consumption increases commensal Clostridium. But if you have a pathogenic Clostridium overgrowth meat consumption will also increase it which increases TMAO levels. TMAO has been linked to cardiovascular disease, diabetes and chronic illness. Most I have met who have IBD-C have health issues that inhibit their ability to consume those things or digest them. What is strange to me and it is something I am just learning about is they all seem to have issues with cyp21a1. There must be a connection but I do not know enough yet to know what it is. The FXR receptor protects us from overgrowth and infection when activated. Also Clostridia produces secondary bile acids which also protect us from infection and overgrowth. A deficiency in vitamin K can lead to thrombosis and strokes the reason for this is the vitamin K produce by Clostridium is used by other gut microbes that prevent thrombosis. I have an update on that, TMAO inhibits cyp21a1. When a person eats meat or just about any animal flesh especially fish who has pathogenic Clostridium overgrowth they will get ill because it increases the pathogenic Clostridium and increases TMAO levels.
Antibiotics, glyphosate , certain medications and processed foods kill the commensal Clostridium. Commensal clostridium keeps us from being harm by pathogenic Clostridium. Excess iron increases most pathogens in the gut including clostridium.

Flavonoids in foods are toxic to the body until microbes like Clostridium orbiscindens, Flavonifractor plautii and Eubacterium ramulus break them down and make them bioavailable to the body. We have to remember those are produced by the plant to protect themselves so our gut microbes have to change them so we reap the benefits from them. If you lack these microbes and take flavoniods like querciten it could make you ill. This means when a person eats foods that are high in certain phenols or phytochemicals they will become ill if they are lacking the commensal Clostridium.

An overgrowth of pathogenic bacteria can cause excess blood clotting and can cause oxygen deprevation in the body. If you are gaining weight but have hypoglycemia these are the microbes you need to restore. Eubacterium Rectale, Clostridium Butyricum, Clostridium Leptum, Faecalibacterium Prausnitzii, Clostridium Cellulose, Eubacterium Cellulosolvens, Coprococcus Eutactus,Coprococcus Comes, Eubacterium Halii, Anaerostipes Caccae, Megaspora spp and all the Roseburia species. These produce short chain fatty acids and will restore B-oxidation. This happens in IC and Crohn’s increasing nitric oxide, taking Saccharomysce boulardii, Bacillus clausii, and decreasing pathogenic Clostridium will correct this. Lactobacillus acidophillus, Lactobacillus rhamnosus, Lactobacillus planatarum and Enterococcus Faeccium also helps to reduce pathogenic Clostridium. Bacillus clausii, and Bacillus boulardii also kills pathogenic Clostridium.

Along with increasing commensal Clostridium like Clostridium Butyricum increasing Lactiobacillus Rueteri keep you from getting pathogenic Clostridium. Most common is Clostridium difficile.

A person should not take high doses of supplements. High dose supplementation with zinc can make a person copper deficient and can cause an overgrowth of pathogenic Clostridium.

Garlic is very effective against Clostridium and other gut pathogens. If you smash it and leave it turn yellow is is even more potent because it increases the levels of allicin. Allicin is sold as a supplement. Garlics effectiveness can be enhances by taking it with cinnamon because cinnamon inhibits pathogenic Clostridium from being able to replicate. Myrrh (Cammiphora myrrha) helps to fight pathogenic Clostridium. Black seed oil has also been found to be effective at inhibiting pathogenic Clostridium growth. Consuming milk products that have been fermented with Lactobacillus Rhamnosus , Lactobacillus paracasei, and Bifidobacteria spp also reduces pathogenic Clostridium.
Streptococcus thermophilus and Bifidobacteria breve help to prevent the diarrhea caused by Clostridium overgrowth. Bifidobateria pseudocatenulati, also prevents Clostridium infection.
Lactobacillus reuterii converts glycerol found in coconut oil and olive oil to a metabolite that enhances the rest of our gut microbes ability to fight pathogenic Clostridium. Mugwort and wormwood reduces candida and Clostridium. Berberine reduce gut pathogens and also reduces gut inflammation. It is found in bayberry, barberry, oregon grape and coptis.

Increasing Butyrate and other short chain fatty acid producers increases resistance to infection, reduces inflammation , and helps to protect the gut. The gut uses butyrate for energy needed for healing. It normalizes the gut bacteria which prevents diarrhea and constipation. Butyrate increases Bifidobacteria and Lactobacillus bacteria. Butyrate inhibits the growth of most pathogenic microbes and prevents H Pylori overgrowth. Butyric acid reduces allergies and reduces histamine production in the body. Butyrate protects nervers and stimulates nerve cell growth in the brain. Butyrate increases fat burning and prevents fatty liver disease.

Many with inflammatory bowel diseases and chronic illness have been shown to be deficient in Clostridium Leptum. Has been shown to help with asthma and allergies.
Bacteroides Thetaiotaomicron secrete natural antibiotics which keep the gut balanced. It is also responsible for innate immunity in the intestines. It reinforces the mucous membrane. It reduces inflammation. It also helps break down carbohydrates. Prevents constipation and improves bile production. But high levels can promote Clostridium infection.
A microbiotia accessable carbohydrate diet increases beneficial bacteria that produce short chain fatty acid. This consist of fiber and carbohydrates. This increases the bacteria that produce short chain fatty acids. If we do not produce enough SCFA we will become constipated and bile production will become inhibited. This will also happen if we develop malabsorption from having high bacteroidetes, methane producing and sulfur digesting bacteria. They contribute to malabsorption, constipation and inhibited bile production from a lack of SCFA in the body. Our blood glucose levels will also become very low and we may develop high insulin levels. Clostridium infection is also a major cause of this and indicates we are lacking the microbiota that protect us from it. This can cause a cycle that is hard to break because bile is needed to prevent clostridium infection.
Clostrium Leptum has been found to be low in those with inflammatory bowel disease and Chohn’s disease. It breaks down carbohydrates and produces butyrate. It reduces inflammation.
Pediococcus pentosaceus prevents clostridium infection of the digestive system by reducing zonulin. This would improve bile production. It also reduces inflammation. It produces natural antibiotics that fight bacteria.
Bacillus Coagulins prevents lactose intolerance. Helps break down food better increasing nutrient absorption. Helps prevent diarrhea. Prevents gas, bloating and intestinal inflammation. Helps prevent constipation. Reduces endotoxins. Improves bladder and vaginal health. Helps keep H Pylori commensal. Yaks have Bacillus velezensis Jt3-1 which is very effective against pathogenic Clostridium infection.
Clostridium Butyricum help prevent many food born pathogens, helps produce short chain fatty acids, it prevents diabetes and helps keep the sulfur digesting and ammonia producing bacteria like Bacteroidetes and Firmicutes from getting too high. If those are high a person can develop diabetes, malabsorption from inhibited bile production and inflammation. It prevents insulin resistance. Clostridium Bytyricum also protect from bartonella.
Faecalibacterium Prausnitzii Faecalibacterium a deficiency can lead to irritable bowel syndrome. Produces butyrate from fiber. A deficiency in F Prausnitzii can lead to asthma, obesity, depression ,Chohn’s disease and inflammatory bowel diseases. A deficiency in this can lead to high TH17 levels resulting in psoriasis. Those with cardiovascular disease and Type 2 diabetes are usually low in F Prausnitzii. Akkermanis Municiphila needs it and vice versa. Prevents inflammation.
It also helps balance the immune system. It needs Akkermansia to thrive. Oligosaccharides increase F. Prausnitzii. It prevents celiac disease. It induces IL-10. It is necessary for fatty acid oxidation. It produces butyrate. It produces Salicyclic acid which is what they make aspirin from. It produces raffinose a type of oligosaccharide that decreases gut permeability which prevents leaky gut. It along with other commensals form biofilms that help protect and heal the gut. Decreases zonulin which preserves the integrity of the tight protein layer. If zonulin levels get too high a person will develop lactose, gluten and gliadin intolerance.

Clostridium Butyricum is a butyrate producing bacteria . Clostridum butyricum kills it’s cousin that cause negative health effects. It also increases IL-10 . It also decreases inflammatory cytokines. There are studies that have shown if the other healthy gut bacteria are not present this one like many of the bacteria species used in probiotics could start causing health problems.It promotes better bowel movements.
E Coli Nissle 1977 doesn’t only protect us from it’s cousins that cause ill health effects it also protects against other types of infections. It has been shown to put inflammatory bowel disorders into remission. It has too many benefits to mention. The only way I know of getting this one is through a probiotic called Multaflor.
Lactobacillus Acidophilus prevents diarrhea, helps protect against clostridium infections. Those with interstitial cystitis are usually deficient in this one. It helps reduce symptoms of inflammatory bowel disorder. Acidophilus regulates our ph of our digestive system and bladder and keeps it at the correct ph. Our bladders and digestive system need to be acidic. An akaline environment can cause a loss of our good gut bacteria and promote the growth of the bad. It can also cause candida to go from commensal to a pathogenic form. Many with interstitial cystitis have found relief using L Acidophilus suppositories. Those who are having difficulty gaining weight usually have excess L acidophilus levels because it speeds up metabolism and those who have problems with weight gain are often deficient in acidophilus.
Bifidobacterium increased the acid levels of our digestive tract which inhibits bad bacteria from growing and help keep our digestive tract acidic. It has been shown to reduce symptoms of irritable bowel disease.Bifidobacterium Infantis protects us from autoimmune disorders by regulating our T cells. B infantis also helps protect our digestive tract.
Lactobacillus Crispatus doesn’t only prevent obesity it prevents candidia infection.
When a person has pathogenic Clostridium overgrowth or Candida overgrowth lipopolysaccharides produces by gut microbes will enter the blood stream which will make the body react the same way you would if you had an infection. If you increase the microbes that reduce LPS it will reduce the symptoms.
There are gut bacteria that reduce LPS, Bifidobacterium, Lactobacillus GG, Saccharramyces Boulardii Bacillus Infantis, B Longhum, Clostridium Butyricum, E Coli Nissle 1917, Lactobacillus Brevis, L Casei, and many others. Fruit pectin reduces LPS. The pectin is the waxy substance found on the skins of many fruits. One of these articles mentions corn flakes. Corn is GMO and causes leaky gut which would make things worse. I do not recommend eating any GMOs they themselves can cause sepsis.
Exercise reduce LPS.
Stimulating the vagus nerve decreases LPS and inflammation.
Once you start taking a probiotic with commensal Clostridium you can start taking polyphenols. Polyphenols reduce LPS. These are usually highest in darker colored fruits and vegetables.
Resistant starch decreases LPS by increasing the good gut bacteria. This should be introduced slowly because when the good gut bacteria increase the bad die off which will cause bloating and herxing. I got very ill when I tried to eat resistant starch. I had to start with small amounts and slowly increase my intake.
Improving protein tolerance
Ursodeoxycholic acid (UCDA) produced by certain microbes in the microbiome activates FXR. Phenylalanine, tyrosine, and leucine also activate FXR receptor. But those who have mitochondrial issues may not be able to properly metabolize them or if a person has too many microbes involved in protein proteolysis they may react to those proteins. Bifidobacterium animalis, Ruminococcus gnavus, Clostridium absonium, and Clostridium buratii produce UCDA.
Ursodeoxycholic acid (UCDA) produced by certain microbes in the microbiome activates FXR. Phenylalanine, tyrosine, and leucine also activate FXR receptor. But those who have mitochondrial issues may not be able to properly metabolize them or if a person has too many microbes involved in protein proteolysis they may react to those proteins. Bifidobacterium animalis, Ruminococcus gnavus, Clostridium absonium, and Clostridium buratii produce UCDA.
These microbes produce urease and increase ammonia levels in the gut and bladder. Clostridium perforingens, H pylori overgrowth, Proteus mirabulis, Salmonella spp, Staphylococcus saprophyticus, ureiaplasma urealyticum yersinia, Eterocolitica, Cryptococcus neoformis, and Coccoides posadii. Many fungal infections can cause increase urease or ammonia levels. Keep in mind that eating too many processed foods, toxins in our foods, metabolic or gene issues can cause high ammonia levels causing the overgrowth of these microbes.
These microbes help fight viral infection including herpes.
Lactobacillus acidophilus, Lactobacillus Brevis, Lactobacillus delbrueckii, Lactobacillus planatarum, Lactobacillus gasseri, Lactobacillus vaginosis, Lactobacillus jensii, Enterococcus Faecium, Clostridium orbiscindens.
Clostridium orbiscindens helps balance hormones.

Reducing oxidative stress
Some things that activate Nrf2 is bile acid ursodeoxycholic acid produced by certain gut microbes. Ruminococcus gnavus, Clostridium baratti, Eubacterium aerofaciens, Lactobacillus planatarum produces ursodeoxycholic acid. Ursodeoxycholic acid improves bile flow, prevents gall stones, reduces inflammation and prevents oxidative stress. Some phytochemical Nrf2 activators are querciten, curcumin, resveratrol, and citrus peel extract.
Butyrate producing microbiota increase TGF-B which is needed to prevent some commensals from becoming harmful. A deficiency in TGF-B can lead to inflammatory bowel disease which can cause bile to be inhibited. The most important of these are F Prausnitzii, Clostridium Leptum, Eubacterium Rectale and Roseburia. If deficient in IL-10 or TGF-B the microbiota that stimulates bile will become pathogenic in nature IL-10 and TGF-B work together to keep them in check so they continue to function properly. I discuss bile in more detail at the end of this article.

There are health problems that can cause sensitivity to Salicylic acid.

Cox-1 inhibition is the cause of the salicylic acid sensitivity. This occurs with many types of infection , it is caused by the loss of Deoxycholic acid producing microbes. High levels Clostridium difficile and low levels of commensal Clostridium is the main cause of salicylate sensitivity. This can also be caused from inhibited sulfation. Metabolic issues in metabolizing phenylalanine can also cause it. Microbes that cause high TMAO levels can cause salicylate sensitivity. If this occurs a person will have high histamines and systemic inflammation that resembles mast cell activation syndrome. To correct this usually addressing the metabolic issues, balancing the gut bacteria and fixing digestive issues will be needed.

Salicylic acid is natural aspirin , but straight aspirin is man made and can irritate the gut.

There is such a variety of information on what food contain salicylate that I could not list them here because each list I found was different.

Symptoms will be similar to high histamines. It is caused by high levels of leukotriens which is usually a result of intestinal inflammation. The inflammation is usually caused by COX-1 inhibition. In many it may be necessary to address sulfation. For example Lyme and Clostridium can inhibit sulfation. So can many toxins like glyphosate and mercury.

The symptoms of salicylate sensitivity will occur after consuming them. If you experience the symptoms below write down the foods and see if they are high in salicylates.

Symptosm of salicylate sensitivity are:

Rosy looking cheeks.

difficulty breathing or asthma like symptoms.

Stuffy nose

gas , bloating or abdominal pain.


hives or skin swelling.

Ringing or buzzing in the ears.

Swelling in lower layer of skin (angiodema)

Tongue or eye swelling.

Frequent headaches

frequent urination

persistent cough


memory loss


psuedoanaphlaxis (throat swelling)

The herbs most potent for reducing leukotriene has side effects so I would not take high dose these are boswellia, and butterbur. Omega 3 oils reduce leukotriene. Omega 6 fatty acids should be avoided except evening primrose. It is a type of Omega 6 that actually reverses the inflammatory effects of other Omega 6s. Antioxidants such as grape seed extract and pine bark extract reduces leukotrienes.

Restoring secondary bile acids is a must.!divAbstract
E Coli Nissle 1977’s cousin can cause many health problems if not kept in check.
Natural products that fight clostrium.

Commensal clostridium keep the pathogenic ones in check.

Why some get sick from E coli and Clostridium and some don’t.

Reducing clostridium scindens level.

Hydrolyzed whey protein will help with the protein defficiency caused.

Benefits clostridium butyrica

If not addressed C diff can become deadly.

High supplementation with zinc can increase C diff.

Glyphosate increases pathogens. Excess iron will cause the same thing to happen.

Pathogenic clostridium produces TMAO.
Reduces clostridium.
I consider these microbes as being the core microbes. They are needed to maintain microbial balance, immune balance and hormone balance in the body. If you are lacking them your hormone levels will be out of balance. Clostridium butyricum, Eubacterium Rectale, Clostridium Rectale, Clostridium Leptum, Faecalibacterium Prausnitzii, Clostridium coccoides, Roseburia spp, Streptococcus thermophillus, lactobacillus acidophilus, streptococcus hansenii, Eubacterium cellulosolvens, Blautia producta.

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